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Restoring Normal Sleep Reduces Amyloid-beta Accumulation in Mouse Model of Alzheimer’s Disease

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Multiple studies in humans and mouse models indicate that sleep disruptions raise the risk of Alzheimer’s disease (AD) by increasing the accumulation of disease-relevant proteins such as amyloid-beta (A-beta) in the brain. In the current study, a team led by researchers at Baylor College of Medicine discovered that, in an animal model of Alzheimer’s disease, restoring normal sleep by returning to normal the activity of the thalamic reticular nucleus (TRN), a brain region involved in maintaining stable sleep, reduced the accumulation of A-beta plaques in the brain.

Source Here: medicalxpress.com

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Research Shows Why Immune Checkpoint Blockade Impedes but Does Not Stop Glioblastoma Progression

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Like locking the door but opening windows, an immune checkpoint therapy temporarily slows glioblastoma (GBM) progression but fails to establish an effective anti-tumor microenvironment and appears to increase molecular interactions inhibiting long-term immune response, according to studies led by a UCLA research team.

Original Post: medicalxpress.com

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Cancer Cells May Promote Metastases and Resistance to Therapy, Depending on Their State

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A type of cell transformation known as EMT enables cancer cells to break away from the tumor and form metastases elsewhere. However, this process does not always take place in full. Researchers at the University of Basel have now been able to show that tumor cells contribute differently to the formation of metastases and the development of therapy resistance, depending on whether they have undergone full or only partial transformation.

Article: medicalxpress.com

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Smart Wound Dressing Provides Suture-free Closure of Surgical Incisions

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“Sutures? That’s practically medieval!”

Original Article: medicalxpress.com

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